Alcohol dependence potentiates substance P/neurokinin-1 receptor signaling in the rat central nucleus of amygdala

Sophia Khom; Thomas  Steinkellner; Thomas S Hnasko; Marisa Roberto

doi:10.1126/sciadv.aaz1050

Alcohol dependence potentiates substance P/neurokinin-1 receptor signaling in the rat central nucleus of amygdala

Sophia Khom (Corresponding author)
, Thomas Steinkellner
, Thomas S Hnasko
, Marisa Roberto (Corresponding author)

Publications: Contribution to journal › Article › Peer Reviewed

Original language	English
Article number	eaaz1050
Number of pages	11
Journal	Science Advances
Volume	6
Issue number	12
DOIs	https://doi.org/10.1126/sciadv.aaz1050
Publication status	Published - 18 Mar 2020
Externally published	Yes

Funding

This work was financially supported by grants from the NIH/NIAAA AA006420, AA013498, AA015566, AA017447, AA027700, AA021491 to M.R. and NIH/NIA K99AG059834 to T.S., the Pearson Center for Alcoholism and Addiction Research and the Austrian Science Fund (FWF Erwin Schrodinger Fellowships J-3942-B30 to S.K and FWF J-3656-B24 to T.S.).

Austrian Fields of Science 2012

301406 Neuropharmacology
301407 Neurophysiology
301405 Neuropathology

Keywords

Adaptation, Physiological
Alcoholism/etiology
Animals
Central Amygdaloid Nucleus/metabolism
Disease Models, Animal
Disease Susceptibility
Gene Expression
Immunohistochemistry
Male
Rats
Receptors, Neurokinin-1/agonists
Signal Transduction/drug effects
Substance P/metabolism
Substance Withdrawal Syndrome
gamma-Aminobutyric Acid/metabolism

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

Access to Document

10.1126/sciadv.aaz1050Licence: CC BY-NC 4.0

Cite this

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title = "Alcohol dependence potentiates substance P/neurokinin-1 receptor signaling in the rat central nucleus of amygdala",

keywords = "Adaptation, Physiological, Alcoholism/etiology, Animals, Central Amygdaloid Nucleus/metabolism, Disease Models, Animal, Disease Susceptibility, Gene Expression, Immunohistochemistry, Male, Rats, Receptors, Neurokinin-1/agonists, Signal Transduction/drug effects, Substance P/metabolism, Substance Withdrawal Syndrome, gamma-Aminobutyric Acid/metabolism",

author = "Sophia Khom and Thomas Steinkellner and Hnasko, \{Thomas S\} and Marisa Roberto",

note = "Publisher Copyright: Copyright {\textcopyright} 2020 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works. Distributed under a Creative Commons Attribution NonCommercial License 4.0 (CC BY-NC).",

year = "2020",

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doi = "10.1126/sciadv.aaz1050",

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AU - Khom, Sophia

AU - Steinkellner, Thomas

AU - Hnasko, Thomas S

AU - Roberto, Marisa

N1 - Publisher Copyright: Copyright © 2020 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works. Distributed under a Creative Commons Attribution NonCommercial License 4.0 (CC BY-NC).

PY - 2020/3/18

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KW - Animals

KW - Central Amygdaloid Nucleus/metabolism

KW - Disease Models, Animal

KW - Disease Susceptibility

KW - Gene Expression

KW - Immunohistochemistry

KW - Male

KW - Rats

KW - Receptors, Neurokinin-1/agonists

KW - Signal Transduction/drug effects

KW - Substance P/metabolism

KW - Substance Withdrawal Syndrome

KW - gamma-Aminobutyric Acid/metabolism

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Alcohol dependence potentiates substance P/neurokinin-1 receptor signaling in the rat central nucleus of amygdala

Funding

Austrian Fields of Science 2012

Keywords

UN SDGs

Access to Document

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