Role of TLR4 in the Modulation of Central Amygdala GABA Transmission by CRF Following Restraint Stress

F. P. Varodayan, S. Khom, R. R. Patel, M. Q. Steinman, D. M. Hedges, C. S. Oleata, G. E. Homanics, M. Roberto, M. Bajo

Publications: Contribution to journalArticlePeer Reviewed

Original languageEnglish
Pages (from-to)642-649
Number of pages8
JournalAlcohol and Alcoholism
Volume53
Issue number6
DOIs
Publication statusPublished - 1 Nov 2018
Externally publishedYes

Funding

Support for this study was provided by National Institute on Alcohol Abuse and Alcoholism (NIH/NIAAA) grants as part of Integrative Neuroscience Initiative on Alcoholism (INIA) Neuroimmune Consortium [U01 AA013498 (M.R.) and U01 AA020889 (G.E.H.)], NIH/NIAAA R01 AA015566 and AA006420 (M.R.), K99 AA025408 (F.P.V.), and T32 AA007456 (R.R.P. and M.Q.S.), as well as the Austrian Science Fund (FWF J-3942 to S.K.) and the Pearson Center for Alcoholism and Addiction Research.

Austrian Fields of Science 2012

  • 301406 Neuropharmacology

Keywords

  • TOLL-LIKE RECEPTOR-4
  • MICE LACKING CD14
  • INDUCED NEUROINFLAMMATION
  • ALCOHOL-DEPENDENCE
  • ETHANOL-CONSUMPTION
  • GABAERGIC TRANSMISSION
  • NOCICEPTIN/ORPHANIN FQ
  • PSYCHIATRIC-DISORDERS
  • GENE-EXPRESSION
  • GLUCOCORTICOIDS

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